P43: Secondary Demyelination in Spinal Cord Injury

p43: secondary demyelination in spinal cord injury

the pathology of spinal cord injury (sci) comprises multiple processes characterized by extensive cell death and glial scar formation, which together limits axonal regeneration. in particular, variations in chronic demyelination of the lesion have been found to be largely independent of injury intensity, suggesting a secondary pathologic origin. this chronic demyelination results in progressive...

Inflammogenesis of Secondary Spinal Cord Injury

Spinal cord injury (SCI) and spinal infarction lead to neurological complications and eventually to paraplegia or quadriplegia. These extremely debilitating conditions are major contributors to morbidity. Our understanding of SCI has certainly increased during the last decade, but remains far from clear. SCI consists of two defined phases: the initial impact causes primary injury, which is foll...

Bradycardia Secondary to Cervical Spinal Cord Injury

Tumefactive demyelination of the spinal cord.

Demyelinating lesions of the brain have been frequently reported to mimic tumors, whereas very few cases of tumefactive demyelination of the spine have been reported. We report a patient with tumefactive demyelination of the spinal cord who was clinically, by MRI, and by frozen section diagnosed with tumor and had complete surgical resection. Hence patients with spinal cord lesions suspicious o...

W5: Animal Models of Spinal Cord Injury

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Natural Polyphenols and Spinal Cord Injury

Polyphenols have been shown to have some of the neuroprotective effects against neurodegenerative diseases. These effects are attributed to a variety of biological activities, including free radical scavenging/antioxidant and anti-inflammatory and anti-apoptotic activities. In this regard, many efforts have been made to study the effects of various well-known dietary polyphenols on spinal cord ...